<?xml version="1.0" encoding="utf-8" standalone="yes"?><rss version="2.0" xmlns:atom="http://www.w3.org/2005/Atom"><channel><title>Alzheimers | Tanigawa Lab</title><link>https://tanigawalab.org/tags/alzheimers/</link><atom:link href="https://tanigawalab.org/tags/alzheimers/index.xml" rel="self" type="application/rss+xml"/><description>Alzheimers</description><generator>Hugo Blox Builder (https://hugoblox.com)</generator><language>en</language><lastBuildDate>Thu, 04 Sep 2025 00:00:00 +0000</lastBuildDate><image><url>https://tanigawalab.org/media/logo_hu_6ec5cb994dd998e6.png</url><title>Alzheimers</title><link>https://tanigawalab.org/tags/alzheimers/</link></image><item><title>Single-cell multiregion epigenomic rewiring in Alzheimer’s disease progression and cognitive resilience</title><link>https://tanigawalab.org/publications/2025/ad-multiregion-epigenomics/</link><pubDate>Thu, 04 Sep 2025 00:00:00 +0000</pubDate><guid>https://tanigawalab.org/publications/2025/ad-multiregion-epigenomics/</guid><description>&lt;p&gt;Alzheimer’s disease (AD) is a neurodegenerative disorder characterized by progressive cognitive decline, yet its epigenetic underpinnings remain elusive.&lt;/p&gt;</description></item><item><title>Human microglial state dynamics in Alzheimer's disease progression</title><link>https://tanigawalab.org/publications/2023/microglia-states/</link><pubDate>Thu, 28 Sep 2023 00:00:00 +0000</pubDate><guid>https://tanigawalab.org/publications/2023/microglia-states/</guid><description>&lt;p&gt;Here we report 194,000 single-nucleus microglial transcriptomes and epigenomes across 443 human subjects and diverse Alzheimer&amp;rsquo;s disease pathological phenotypes.&lt;/p&gt;</description></item></channel></rss>